I posted this in another forum, but since the person who dug up this old thread to talk about "gene 6" in multiple forums... In case anyone wants some information about "gene 6"...better/correctly known as "P6"...as it pertains to current discussion based on a study by the EFSA... This is a very wide range of proteins found in virus encoding from HIV to mosaic virus...these proteins are also found in the smoke of burning meat and tobacco. It's a very wide range. In this case, one of the biggest dangers would be a chance encoding to re-invigorate the "dead" version of cauliflower mosaic virus (or P6 residues) that's very commonly used as a carrier string for DNA/RNA insertion that it's inserted into. This could lead to some allergy problems, too, even if it doesn't fully express the mosaic virus but still overlaps enough to express P6 proteins. P6 is a known allergen, though it's not one that everyone is sensitive to. The expression of this gene is highly unlikely, though...and would be regulated to a single (or very small groups) of plants doing this replication rather than entire seed source or a field suddenly replicating mosaic virus or P6 residues. If it is the case that encoding suddenly made it large-scale available it would show up heavily in the research stage and it wouldn't make it out into the consumer market since it's showing inferior/bad genetic expression. One of the biggest parts of GMO research is tossing out 99%+ of everything you're actually trying to create because positive effects of expression aren't stable enough to sell it as seed...or it's showing "bad" expressions. There's a lot of otherwise harmful viruses (to plants or humans) used to insert GMO traits for start/end points into a genetic change that are made inert (and distinctly different) from their original genetic package, but still contain large parts of what makes up the virus, itself. Viruses can easily carry genetic information and they're ideal vehicles for transferring it. The genetic carriers of the virus are merely vehicles. Once you change the "genetic package" inside a virus it's not even what you started with. The "guts" are changed dramatically. If you put a Dodge Neon engine in a Porsche very few people would still consider it a Porsche. That's the level of dramatic change in sequencing going on inside of these packages. You can take certain virus types, depending on what you're trying to achieve, and precisely insert genetic information with start/termination points into existing DNA/RNA...totally turning it's genetic information into something totally different in both makeup and application. Btw, to those with P6 protein sensitivities...this would be a big deal. I'm not trying to knock the research at all. I'm just saying it's overlapping expression would most likely be contained to a very few plants in a field, not widespread. While genetic start/termination points are very good with insertion and replication once stable, nothing is perfect when you're exchanging genes...we see it even natural breeding. The major problem with this particular chain of insertion is the overlapping of the 2 sequences given as example in the paper and what could happen as a consequence of them being genetically linked so closely together...even if there's a very small chance of it happening as defined. It's also worth mentioning we're talking a single virus carrier, not the 100s of types (or the 20-ish most commonly used) carriers. It would also be greatly influenced by the new information inserted, what was cut out, and where the start/termination points overlap (if there is any replication overlap). There's more than 1 way to insert genetic information into virus and the chances of overlap encoding or reversion is different depending on the type of method used.

I was the oldest of four children and the last one pregnant. I think my parents had just about given up on a grand baby from me. When I finally got pregnant (at 35), I gave them a Christmas card from our baby due the following August! I had tears as Mom opened the card.

My DS was 6-7 and home from school sick in bed. We had a snow and everything was covered and freezing cold outside. DS called me into the bedroom and told me to look out at the bird on the telephone wire. The poor thing looked like he was frozen to the wire, but apparently not because after a while as we watched, he appeared to fall to the ground. DS asked me to go see what was wrong with the bird. So of course I bundled up, put on boots and gloves and went out on rescue. I brought the bird in and we could see that he had a grain of corn stuck in his throat. I managed to dislodge it and he was then ready to go. I let him out the door and he flew away. I've forgotten many of the things DS did to aggravate me, but I still remember that day when he had to know that one bird was helped. He is still like that, very caring. Sue

I have a wild kingdom going..and I would think Crows would make fast work of Mantids. I used to see Jays and Mockingbirds..but once cats started in this neighborhood,rare to see them. Possums would make a snack of them...they eat anything. Even the number of fence lizards has gone down..when they had showed up for a few years out of nowhere. Same for Garter Snakes come to think of it. Could be the wet weather 2 years in a row spread everybody out?