Rose Mosaic Virus - Rooting
mark_roeder 4B NE Iowa
2 years ago
last modified: 2 years ago
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Rose Mosaic Virus & bloom quality
Comments (5)Yes, probably most body bag roses are infected. A lot of that has to do with the fact that the root stock of what ever mother plant the buds were taken from was infected and they were probably grafted to infected root stock as well. As for the difference in your blooms I doubt it had to do with RMV but more likely was due to the all around inferior quality of the plant. Most of those body bags are not grade #1 but more likely #1 1/2 or #2 grade. Depending on where it was from it may have been badly grafted, was certainly badly handled and pretty well whacked off for packaging. Not an auspicious start for any rose. I have many virused roses in my garden and have never noticed any difference in their over all vigor. For the most part they rarely even show symptoms except in times of great stress. Hmmm...like maybe this awful winter! We'll see... We have this discussion about RMV routinely here and there are two opposing sides. Listen to them both and make your own decision as to what you're willing to except. There are some roses out there that can not be had any other way then virused. They were more or less bred/introduced already virused since the only way they were ever propagated was to be grafted to virused root stock. Being that Proud Land was introduced in 1969 it may well be one of those. There have been strides taken to get more varieties cleaned up but it's a long road and will take some time yet before it's done. And we may lose some varieties altogether in the process. In our already shrinking rose world I hate to see that happen....See Morepilgrim and rose mosaic virus
Comments (11)windeaux, thank you for providing your experimental details. I do not have any virused roses to experiment with. (I am now shutting down my 1000 rose garden due to age and health.) From the literature I would expect that cuttings taken in your zone in August (highest possble temperature) would have the greatest chance of being virus free. Also it appears that old wood may be preferable to new wood. Also, it probably would be advantageous to take cuttings from branches that did not have virus symptoms in the spring (in fact I would recommend that virused branches be removed from the plant when first observed). The first paper that drew my attention to this behavior is (please, keep in mind the reversal of our seasons with theirs): Authors: Dal Zotto, A.; Nome, S. F.; Di Rienzo, J. A.; Docampo, D. M. Author Address: Instituto de Fitopatologia y Fisiologia Vegetal INTA, 5009, Cordoba, Argentina. Title: Fluctuations of Prunus necrotic ringspot virus (PNRSV) at various phenological stages in peach cultivars. Published in: Plant Disease, volume 83, pages 1055-1057, (1999). Abstract: " Fluctuations in Prunus necrotic ringspot virus (PNRSV) concentration were researched in single plants of six peach (Prunus persicae) cultivars-Kurakata, Red Haven, Nectar Red, Start Delicious, Meadowlark, and Loadel-by double antibody sandwich-enzyme-linked immunosorbent assay (DAS-ELISA) of dormant buds (May, June), flowers (September), new sprouts (November), and mature leaves (January) (Southern Hemisphere). The optimum extract dilution (sample weight per buffer volume) to detect the virus was also quantified. The average absorbance patterns of the six cultivars show a steady increase in virus concentration, ranging from A405nm 0.61 in May to A405nm 0.86 in July for dormant buds, to A405nm 1.22 in September in flowers, to 1.53 in November in new sprouts, where the highest concentration was found. Virus concentrations in mature leaves drop to values similar to those of noninfected plants in January (A405nm 0.12). The yearly average (six noninfected peach trees) ranged from A405nm 0.04 to A405nm 0.08. This drop coincides with an increase in summer temperature and attenuates foliation symptoms caused by PNRSV. Analysis of dormant buds, flowers, or new sprouts with 5-cm-long leaves was reliable to differentiate infected from noninfected plants. Cluster analysis of absorbance profiles for single plants of cvs. Loadel and Meadowlark, however, showed a comparatively low profile, with a drop at flowering time (A405nm 0.20 in September) close to the average of healthy controls. The difference between infected and healthy plants did not become apparent in all cultivars from the analysis of plants at a given phenological stage, for example by the analysis of flower only, the material most preferred to diagnose the virus. Therefore, plants should be analyzed during flowering and sprouting or flowering and dormancy (dormant buds)." Here is a link that might be useful: peach southern hemisphere study...See MoreQuestion about Rose Mosaic Virus
Comments (51)comtessedelacouche (10b S.Australia: hotdryMedclimate) I started each post to you with the following qualifier: "since you are from Australia, you may find these links of use:" Please notice the use of "you may find". I suggest that if you find something in the links that you are interested in obtaining addition information / viewpoints about, that you start a new thread on that point. If your rose growing skills are anything like what I saw illustrated in the annual, congratulations!...See Morerose root to root of a nearby rose grafting and virus transfer
Comments (12)Nature is complex. Apparently mechanical transmission is not always successful. I wonder if the mites contribute a cofactor that helps the virus overcome the immune system. The following may apply to this point: "but no consistent method of mechanical inoculation has been developed for both wheat and maize that can satisfactorily bypass the use of the wheat curl mite as a vector (Mirabile, 2001; Seifers et al., 1997; Skare et al., 2003)." http://amarillo-tamu-edu.wpengine.netdna-cdn.com/files/2010/12/A-new-eriophyid-mite-borne-membrane-enveloped-virus-like-complex-isolated-from-plants.pdf ---------------------------------------------- "We are beginning to determine what proteins are present in the mite saliva, in order to determine how the mites are suppressing the immune reactions. In collaboration with Doug Cavener and Kaori Iida, we have isolated the genes for several genes involved in honeybee immunity. Not all invaders are met with a well-defined immune response, as during disease transmission by insect vectors. Understanding the mechanisms that underlie co-evolution between the vector and disease organism is critical to understanding how the disease transmission cycle might be disrupted. Through interdisciplinary collaboration with Drs. F. Gildow (PSU) and S. Gray (Cornell), we are examining the mechanisms underlying virus/vector specificity in aphids at the accessory salivary gland, the primary site determining transmission of different species of luteoviruses. In my lab, we have identified several proteins that bind to luteovirus in a vector-specific manner and that are also recognized by antibodies that mimic the virus. We are isolating the genes encoding these proteins for studies of the evolution of these virus/vector interactions, among different aphid and viral species. This research may further understanding of mechanisms underlying the vector-specificity of mosquitoes for transmission of alphaviruses." http://ento.psu.edu/directory/dxc12...See Moremark_roeder 4B NE Iowa
2 years agolast modified: 2 years agoValRose PNW Wa 8a
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2 years agolast modified: 2 years agomark_roeder 4B NE Iowa
2 years agolast modified: 2 years agoHenry Kuska
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mark_roeder 4B NE IowaOriginal Author